As a result, the heart-specifically its right side-must pump harder than usual to keep up with the body’s needs. The increased blood pressure makes it harder for the heart to pump blood to the lungs. (Note that pulmonary hypertension refers only to the blood pressure in the pulmonary arteries it is different than the blood pressure that health care providers measure with a blood pressure cuff around your upper arm.) In a person with CTEPH, the narrowing of the pulmonary arteries causes the blood pressure in them to rise-called pulmonary hypertension. The oxygenated blood then returns to the heart, which pumps it to the rest of the body. In a healthy individual, they deliver blood from the heart to the lungs, picking up oxygen along the way. The pulmonary arteries play an essential role in blood circulation. The chronic blood clots cause scarring and narrowing of the pulmonary arteries, resulting in a reduction of blood flow through-and an increase in blood pressure in-these blood vessels. In rare cases, the blood clots from pulmonary embolisms remain intact in the pulmonary arteries for a long time.

Although these blood clots often break apart on their own, in some cases an acute pulmonary embolism requires treatment, such as blood thinners or clot-busting drugs, to dissolve the clots and restore normal blood flow.

It starts with an acute pulmonary embolism, which is a blockage in a pulmonary artery, usually from a blood clot. CTEPH is a rare condition in which long-term-or chronic-blood clots form in the lungs’ arteries.

Clot buster medicine